अध्ययन भन्छः धूमपानले दिमाग पनि खियाउँछ
A mere handful of studies indicate that chronic cigarette smoking by itself has adverse effects on brain structure and cognitive functioning. However, to date, none is not aware of any published studies using magnetic resonance imaging the brain damage found in alcoholics in treatment is entirely from chronic excessive alcohol consumption or from being smokers, as well.
But this recent study done among smokers and drunker results indicate that chronic cigarette smoking increases the severity of brain damage associated with alcohol dependence.That is, the combined effects of alcohol dependence and chronic smoking are associated with greater regional brain damage than chronic alcoholic drinking or smoking alone. Their studies show that this exacerbation of the alcohol-induced brain damage is most prominent in the frontal lobes of individuals studied early in treatment.
But this recent study done among smokers and drunker results indicate that chronic cigarette smoking increases the severity of brain damage associated with alcohol dependence.That is, the combined effects of alcohol dependence and chronic smoking are associated with greater regional brain damage than chronic alcoholic drinking or smoking alone. Their studies show that this exacerbation of the alcohol-induced brain damage is most prominent in the frontal lobes of individuals studied early in treatment.
The damage to the brain’s frontal lobes could compromise the success of treatment and recovery. That part of the brain is responsible for multiple functions of everyday life, including a person’s ability to accurately judge or anticipate the consequences of their actions. The study found that cigarette smoking alone caused damage to neuronal viability and cell membranes in the mid brain and on cell membranes of the cerebellar vermis. These brain regions are involved in fine and gross motor functions and balance and coordination.They also observed that higher smoking severity among smoking recovering alcoholics was associated with lower N-acetylaspartate levels in lenticular nuclei and thalamus, areas also involved in motor functions.